As program coordinator and evaluator for the COBRE, my role is to coordinate the administrative activities of the COBRE as well as to help establish benchmarks of success for the COBRE and monitor our success in reaching them. While this is my first experience in an administrative position, my understanding of what comprises a successful research program is based on extensive research experience. I earned my Ph.D. in Neuroscience from Tufts University in August 2010. My graduate work focused on the auditory system, exploring how stress hormones modulate hearing sensitivity and susceptibility to noise-induced hearing loss. I then spent two years as a postdoctoral fellow at the USDA Jean Mayer Human Nutrition Research Center on Aging (HNRCA) where I studied fat metabolism and the inflammatory pathways linking obesity to metabolic disease. I look forward to using my knowledge of scientific research and the research community to help build the COBRE for the Neurobiology of Chronic Pain at UNE and contribute to the progress that is already underway in making UNE a competitive and well-recognized research institute in the Northeast.
Sackler School of Biomedical Sciences, Tufts University
The University of Chicago
Basappa J, Graham CE, Turcan S, Vetter DE. The cochlea as an independent neuroendocrine organ: expression and possible roles of a local hypothalamic-pituitary-adrenal axis-equivalent signaling system. Hear Res. June 2012. 288(1-2): 3-18. Review.
Greenberg AS, Kraemer FB, Soni KG, Jedrychowski MP, Yan QW, Graham CE, Bowman TA, Mansur A. Lipid droplet meets a mitochondrial protein to regulate adipocyte lipolysis. EMBO J. Nov 2, 2011. 30(21): 4337-9.
Graham CE, Basappa J, Turcan S, Vetter DE. The cochlear CRF signaling systems and their mechanisms of action in modulating cochlear sensitivity and protection against trauma. Mol Neurobiol. Dec. 2011. 44(3): 383-406. Review.
Graham CE, Vetter DE. The mouse cochlea expresses a local hypothalamic-pituitary-adrenal equivalent signaling system and requires corticotropin-releasing factor receptor 1 to establish normal hair cell innervation and cochlear sensitivity. J Neurosci. Jan. 26, 2011. 31(4): 1267-78.
Graham CE, Basappa J, Vetter DE. A corticotropin-releasing factor system expressed in the cochlea modulates hearing sensitivity and protects against noise-induced hearing loss. Neurobiol. Dis. May 2010. 38(2): 246-58.